Release of the brake of hair growth
Activity of 5α-reductase that converts testosterone to dihydrotestosterone (DHT) is genetically determined in males. DHT contributes to hair loss by inhibiting the production of an important hair growth factor insulin-like growth factor-I (IGF-I), thereby inducing androgenetic alopecia (AGA). IGF-I is an accelerator for hair growth, suggesting that DHT may play a role as the brake of hair growth. Through years of painstaking research, JHGC has developed hair growth theory and knowledge for the suppression of DHT production. Thus, by releasing the brake of hair growth using JHGC’s technique, we were able to restore the normal hair cycle, thereby regaining the normal hair growth.
Stepping on the accelerator of hair growth
IGF-I promotes the hair growth by prolonging the growth phase (anagen) and by shortening the retraction phase (catagen) in hair cycle. JHGC treatments increase production of IGF- 1at the hair follicle, thereby promoting hair growth. This new treatment means the stepping on the accelerator of hair growth and, thus, it enables us to improve alopecia more effectively and efficiently than treatments previously done. By utilizing this dual approach, JHGC has realized the next-generation of hair growth strategy and yield even more robust effects.
Normalization of the scalp condition
Other reasons for hair loss can be: (1) excess secretion of sebum; (2) low blood circulation. Excessive sebum, upon contact with air, is oxidized and turns into oxidized sebum that obstructs the hair pores.
Overproduction of oil Poor circulation arrow With the
correct care,
normal hair
growth be archieved.
The accumulated fat caught in the pores induces inflammation, leading to hair loss.
If blood circulation at the hair follicle is not enough, nutrients are not carried to the hair. Consequently, the hair becomes thinner and easily falls out. We are able to restore the normal hair growth and reduce hair loss under the correct care to the scalp leading to normalization of the scalp condition.
Mechanism of IGF-1 for hair growth effect in scalp. Hair matrix cell
Sensory neurons that transmit the pain and heat sensation to the brain perceive the stimulatory information, thereby releasing CGRP (calcitonin gene-related peptide). Dermal papilla cells increase IGF-I production by receiving CGRP, and IGF-1 thus formed activates keratinocytes to increase its proliferation. Consequently, hair growth is promoted.
