Avocado oil

[Itsthatguy]

Yours is virgin avocado oil?
I'm looking for extra virgin... supposed to have higher smoke point. So can cook / fry

Maybe you can check out Mantova smoke point at 450F. Not extra virgin.

 

[buttbERry]

for me is a no

 

[ahnyaahnya]

I’m using this. Trying to stock up two extra bottles at home when there’s an offer. But cutting down the usage of oil by having more steamed dishes instead.

https://www.fairprice.com.sg/product/grove-extra-virgin-avocado-oil-original-250ml-12620928

Thanks.
I saw that before.
Problem is that it contains garlic.
I can't take garlic... cos of my reflux..unless it's infused.
But maybe , if I can't find anything suitable, I'll give it a try

 

[ahnyaahnya]

What is your current omega-3 to omega-6 ratio? If it exceeds 5, 10, or even 25, further increasing omega-6 intake will only exacerbate systemic inflammation and metabolic dysfunction. The smoke point of an oil is a poor predictor of its suitability for cooking, the key parameter to evaluate is its oxidative stability, often measured by the Oxidative Stability Index (OSI), which reflects its resistance to lipid peroxidation and the formation

Very chim.
I don't know my Omega ratio.
Got test for that?
I do take Omega 3 supplement, 3x 1200 mg daily.

---

Don't understand the rest of what you stated.
I have inflammation problem... arthritis ( bad recently), and Sjogren.

Oh thanks. I just looked it up.
So avocado oil is out for me cos of the high Omega 6.
What about the fruit? Also high Omega 6?

 

[thwysg]

I don't know my ratio

Thanks.
I saw that before.
Problem is that it contains garlic.
I can't take garlic... cos of my reflux..unless it's infused.
But maybe , if I can't find anything suitable, I'll give it a try

Did you get the right one? There’s three different ones - original, garlic and lime. I always use original.

https://www.fairprice.com.sg/brand/grove

 

[KPO_SAHM]

Does anyone here use avocado oil ? How do you store it?

I am using. High melting point. The oil is super exp. I would stock up more if there is offer.
Just store at room temp. But PLS read the ingredient list before making purchase cause there is refined (chemically processed) avocado oil in the market.

 

[KPO_SAHM]

Almost all avocado oils, like olive oils, are fake, so store them like any RBD oil, as they are usually made from low-quality refined seed oils such as canola or soy.

Some olive n avocado oil have triangle HPB logo...better to check the ingredients list than trusting the logo. Due to marketing, consumer would be mislead by them as some aare refined oil (chemically processed)

 

[ahnyaahnya]

What is your current omega-3 to omega-6 ratio? If it exceeds 5, 10, or even 25, further increasing omega-6 intake will only exacerbate systemic inflammation and metabolic dysfunction. The smoke point of an oil is a poor predictor of its suitability for cooking, the key parameter to evaluate is its oxidative stability, often measured by the Oxidative Stability Index (OSI), which reflects its resistance to lipid peroxidation and the formation

So coconut oil is better?
Bought it many years ago but didn't dare use it cos people say bad for cholesterol.
But I recently read somewhere online that although it does raise cholesterol levels, it raises HDL, big cell LDL . But reduces small cell LDL ( the bad part of LDL)
Dunno true or not.

 

[Apparatus]

Does anyone here use avocado oil ? How do you store it?

I bought a EV Cold Pressed Avogado oil in dark glass bottle and keep it away from direct sunlight on the countertop

 

[ahnyaahnya]

I bought a EV Cold Pressed Avogado oil in dark glass bottle and keep it away from direct sunlight on the countertop

Isn't our ambient temperature too high?
You can keep for how long?

 

[Apparatus]

Isn't our ambient temperature too high?
You can keep for how long?

There's a shelf life indicated on the bottle mah. Or can just keep it in the cabinet away from direct sunlight can liao

When you use it if it smells/tastes bad or the color turns bad then throw it away lor

 

[Checkyrmed]

So coconut oil is better?
Bought it many years ago but didn't dare use it cos people say bad for cholesterol.
But I recently read somewhere online that although it does raise cholesterol levels, it raises HDL, big cell LDL . But reduces small cell LDL ( the bad part of LDL)
Dunno true or not.

Current clinical guidelines set relatively strict targets for controlling LDL cholesterol, which means that many individuals, even when their LDL levels are already near the lower range, are still advised to take cholesterol-lowering medications. At the same time, the protective role of HDL cholesterol is often not given sufficient attention.

Evidence from research shows that relying solely on LDL as the basis for risk assessment may not be comprehensive. Some independent researchers argue that using total cholesterol to HDL ratios, LDL to HDL ratios, or non-HDL cholesterol as markers can more accurately reflect cardiovascular risk and may provide greater clinical value than focusing on LDL alone.

It is worth noting that the European Society of Cardiology and a number of European cardiologists have already recommended incorporating non-HDL cholesterol and ApoB into clinical practice as risk assessment and treatment targets, to make up for the shortcomings of relying only on LDL. In the United States, however, guidelines still emphasize LDL reduction as the primary treatment goal, and the use of ratios or ApoB in practice is far less common. This highlights a clear difference between European and American approaches to cardiovascular risk assessment.

The main reason the U.S. continues to emphasize LDL is that nearly all large-scale randomized controlled trials have used LDL-C reduction as the primary endpoint and treatment target, thereby reinforcing LDL’s role within the framework of evidence-based medicine. However, with the wider availability of ApoB testing and growing research showing its superiority in predicting cardiovascular risk, it is likely that the U.S. will eventually incorporate ApoB or non-HDL cholesterol more broadly into clinical practice.

On this point, there is a clear difference in attitude between American and European doctors:

	U.S. Approach	European Approach
Primary focus	LDL-C (“bad cholesterol”)	Non-HDL cholesterol, ApoB, ratios (e.g., total cholesterol/HDL)
Basis for prescribing medication	Whether LDL meets the target	A more comprehensive risk assessment, not just LDL
Guideline stance	Still centered on LDL, because most clinical trials use LDL as the benchmark	Increasingly emphasizes ApoB and non-HDL, viewing them as more accurate
Attention to HDL (“good cholesterol”)	Relatively little emphasis	More consideration of HDL’s protective role
Future trend	May gradually introduce ApoB and non-HDL measures	Already promoting these markers in guidelines

The U.S. approach is relatively narrow, focusing mainly on LDL to decide whether to prescribe medication, while Europe takes a more comprehensive view, incorporating HDL, non-HDL, and ApoB into the assessment. As ApoB testing becomes more widely available, the U.S. may eventually follow suit.

Peter went for a medical checkup, and his report showed that his LDL (commonly known as “bad cholesterol”) was slightly elevated. The doctor looked at the report and immediately suggested cholesterol-lowering medication. Strangely, however, Peter’s HDL (“good cholesterol”) level was quite good, yet nobody mentioned it. Peter wondered: is heart health really judged only by LDL?

In reality, the answer is not that simple. In the United States, doctors often set very low targets for LDL. Even if your LDL is already close to the normal range, you may still be advised to take medication. At the same time, the protective role of HDL is frequently overlooked.

Some studies have found that looking only at LDL does not fully reflect the risk of heart disease. A more meaningful approach is to consider HDL at the same time, or to use more comprehensive markers, such as the “total cholesterol-to-HDL ratio,” the “LDL-to-HDL ratio,” or “non-HDL cholesterol.”

 

[ahnyaahnya]

Current clinical guidelines set relatively strict targets for controlling LDL cholesterol, which means that many individuals, even when their LDL levels are already near the lower range, are still advised to take cholesterol-lowering medications. At the same time, the protective role of HDL cholesterol is often not given sufficient attention.

Evidence from research shows that relying solely on LDL as the basis for risk assessment may not be comprehensive. Some independent researchers argue that using total cholesterol to HDL ratios, LDL to HDL ratios, or non-HDL cholesterol as markers can more accurately reflect cardiovascular risk and may provide greater clinical value than focusing on LDL alone.

It is worth noting that the European Society of Cardiology and a number of European cardiologists have already recommended incorporating non-HDL cholesterol and ApoB into clinical practice as risk assessment and treatment targets, to make up for the shortcomings of relying only on LDL. In the United States, however, guidelines still emphasize LDL reduction as the primary treatment goal, and the use of ratios or ApoB in practice is far less common. This highlights a clear difference between European and American approaches to cardiovascular risk assessment.

The main reason the U.S. continues to emphasize LDL is that nearly all large-scale randomized controlled trials have used LDL-C reduction as the primary endpoint and treatment target, thereby reinforcing LDL’s role within the framework of evidence-based medicine. However, with the wider availability of ApoB testing and growing research showing its superiority in predicting cardiovascular risk, it is likely that the U.S. will eventually incorporate ApoB or non-HDL cholesterol more broadly into clinical practice.

On this point, there is a clear difference in attitude between American and European doctors:

	U.S. Approach	European Approach
Primary focus	LDL-C (“bad cholesterol”)	Non-HDL cholesterol, ApoB, ratios (e.g., total cholesterol/HDL)
Basis for prescribing medication	Whether LDL meets the target	A more comprehensive risk assessment, not just LDL
Guideline stance	Still centered on LDL, because most clinical trials use LDL as the benchmark	Increasingly emphasizes ApoB and non-HDL, viewing them as more accurate
Attention to HDL (“good cholesterol”)	Relatively little emphasis	More consideration of HDL’s protective role
Future trend	May gradually introduce ApoB and non-HDL measures	Already promoting these markers in guidelines

The U.S. approach is relatively narrow, focusing mainly on LDL to decide whether to prescribe medication, while Europe takes a more comprehensive view, incorporating HDL, non-HDL, and ApoB into the assessment. As ApoB testing becomes more widely available, the U.S. may eventually follow suit.

Peter went for a medical checkup, and his report showed that his LDL (commonly known as “bad cholesterol”) was slightly elevated. The doctor looked at the report and immediately suggested cholesterol-lowering medication. Strangely, however, Peter’s HDL (“good cholesterol”) level was quite good, yet nobody mentioned it. Peter wondered: is heart health really judged only by LDL?

In reality, the answer is not that simple. In the United States, doctors often set very low targets for LDL. Even if your LDL is already close to the normal range, you may still be advised to take medication. At the same time, the protective role of HDL is frequently overlooked.

Some studies have found that looking only at LDL does not fully reflect the risk of heart disease. A more meaningful approach is to consider HDL at the same time, or to use more comprehensive markers, such as the “total cholesterol-to-HDL ratio,” the “LDL-to-HDL ratio,” or “non-HDL cholesterol.”

I was prescribed atorvastin 3 years + ago... 40 mg. Cos LDL was high. I didn't take it. My ratios were good. Over next few visits, got reduced gradually to 10 mg cos I had told doctors I won't take such high dose. In the end, I still didn't take it.
However subsequently ivdid go to GP to do blood test including ApoB. ApoB was too high.
Any idea how to reduce that?
I did read somewhere online a claim that coconut oil reduces the bad part of LDL. Dunno whether true or not.

 

[Checkyrmed]

I was prescribed atorvastin 3 years + ago... 40 mg. Cos LDL was high. I didn't take it. My ratios were good. Over next few visits, got reduced gradually to 10 mg cos I had told doctors I won't take such high dose. In the end, I still didn't take it.
However subsequently ivdid go to GP to do blood test including ApoB. ApoB was too high.
Any idea how to reduce that?
I did read somewhere online a claim that coconut oil reduces the bad part of LDL. Dunno whether true or not.

You did the right thing by going beyond LDL and checking ApoB, since ApoB reflects the number of atherogenic lipoprotein particles (VLDL and LDL) circulating in your blood. High ApoB is much more strongly linked to cardiovascular risk than LDL-C alone.

One of the main drivers of elevated ApoB is insulin resistance. When the body becomes resistant to insulin, the liver produces more VLDL particles which later convert into small dense LDL (Pattern B). This combination of high ApoB, high triglycerides and Pattern B LDL represents the most dangerous lipid profile.

Coconut oil and its primary fatty acid lauric acid are especially relevant here. Lauric acid enters mitochondria without requiring carnitine transport which allows faster fat oxidation and energy turnover. This prevents toxic lipid intermediates such as diacylglycerol and ceramides from accumulating and blocking insulin signaling. By supporting insulin sensitivity lauric acid can indirectly reduce ApoB levels.

Coconut oil also raises HDL and HDL itself helps improve insulin sensitivity in several ways. It reduces chronic low-grade inflammation which drives insulin resistance. It enhances glucose uptake in skeletal muscle. It protects pancreatic beta cells which helps maintain healthy insulin secretion.

Another important point is that while coconut oil raises both LDL and HDL cholesterol in some people, in others LDL actually decreases. What remains consistent is a more pronounced rise in HDL, which improves cholesterol ratios that carry greater weight for cardiovascular risk. At the same time, coconut oil can reduce triglycerides, enhancing VLDL clearance and lowering the burden of ApoB-containing particles. Taken together, this means that despite variations in LDL-C, the overall impact on ApoB may be neutral or even beneficial, depending on an individual’s metabolic profile.

In addition, coconut oil consumption promotes satiety more effectively than long-chain fats which supports weight control. It reduces inflammatory signaling pathways such as NF-κB and TNF-α that interfere with insulin action. It also increases GLUT4 translocation in muscle and fat cells which improves glucose uptake and utilization. All of these mechanisms work together to improve insulin sensitivity and lipid handling which leads to lower ApoB and a healthier cholesterol profile.

Replacing refined omega-6 seed oils such as corn and soy with coconut oil is therefore a practical step to support cholesterol balance and metabolic health. It is not the only factor that matters but it is one of the most powerful and overlooked changes you can make.